Stanozolol / Winstrol builds a solid, high-quality muscle mass without worrying about excessive subcutaneous fluid retention. This makes it a great steroid for “clearing” cycles when water and fat retention are the major problem. It is also very popular among athletes combining it in sports for strength / speed (endurance). Stanozolol / Winstrol helps reduce the amount of binding globulin (SHBG) in the body and this action is much stronger than other anabolic steroids. SHBG has the potential to bind to other steroids, making them useless. This means that taking too many steroids becomes useless because SHBG neutralizes them.
Because Stanozolol / Winstrol reduces the amount of SHBG in the body, it makes it possible for steroids to be taken in a cycle without having to lower the doses. It can be combined with other steroids to help achieve solid and embossed muscles.
When used by men for purposes of physical development, this drug is usually used in the range of 50-100 milligrams per day taken in cycles lasting no more than six to eight weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in muscle mass and muscle density. This agent is most commonly used as an over-the-counter steroid for pre-competition bodybuilding purposes and is not regarded as an ideal bulking agent due to lack of estrogenicity. Women often use 10-20mg per day, which is taken in cycles lasting no more than four to six weeks. The effect of virilization is unlikely at this level of use, although it can not be excluded.
Stanozolol / Winstrol is not flavored by the body and is not measurable estrogenic. Anti-estrogen is not needed when using this steroid because gynecomastia should not even refer to sensitive individuals. Because estrogen is a common culprit for water retention, this steroid instead enhances the formation of pure muscle mass without water. This makes it a beneficial steroid to use during the embossing cycle when retention of water and fat is a major problem.
Although Stanozolol / Winstrol is classified as an anabolic steroid, androgenic side effects are still possible with this substance. These may include attacks of oily skin, acne. Doses higher than those ordinarily prescribed are more likely to cause such side effects. Anabolic-androgenic steroids (AAS) may also increase hair loss in men. Additionally, women are alerted to the potential virilizing effects of AAS. These may include voice thickening, menstrual problems, changes in skin texture, facial hair and enlargement of the clitoris.
Toxicity of the liver:
Stanozolol / Winstrol is a C17-alpha alkylated compound. This change prevents the drug from deactivating from the liver, which allows a very high rate of entry of the drug into the bloodstream after oral administration. The C17-alpha-alkylated AAS may be hepatotoxic. Prolonged or high dose may cause liver damage. In rare cases, life-threatening dysfunction may develop. It is advised to periodically visit a physician during each cycle to monitor liver function and overall health. The administration of C17-alpha-alkylated steroids is usually limited to six to eight weeks in an attempt to avoid the escalating liver strain.
Anabolic-androgenic steroids can have a detrimental effect on serum cholesterol, which increases the risk of atherosclerosis. Consideration should also be given to the fact that both viral types are associated with adverse changes in HDL / LDL cholesterol levels. This side effect is common in anabolic cycles. It can become a health problem when the dose or duration of intake exceeds normal. The oral version should have a bigger “stroke” on cholesterol than the injection because of the sucking method, and the worse choice of both for this side effect.
All AAS, when taken at sufficient doses to increase muscle mass, are expected to suppress endogenous testosterone production. Without interfering with testosterone-stimulating agents, testosterone levels should be normalized within one to four months after discontinuation of treatment. Note that prolonged hypogonadotrophic hypogonadism may develop secondarily from steroid abuse, which requires medical intervention.